"Radiation Chimera" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An organism whose body contains cell populations of different genotypes as a result of the TRANSPLANTATION of donor cells after sufficient ionizing radiation to destroy the mature recipient's cells which would otherwise reject the donor cells.
| Descriptor ID |
D011828
|
| MeSH Number(s) |
B05.200.750.760 G12.470.500
|
| Concept/Terms |
Radiation Chimera- Radiation Chimera
- Chimera, Radiation
- Chimeras, Radiation
- Radiation Chimeras
|
Below are MeSH descriptors whose meaning is more general than "Radiation Chimera".
Below are MeSH descriptors whose meaning is more specific than "Radiation Chimera".
This graph shows the total number of publications written about "Radiation Chimera" by people in this website by year, and whether "Radiation Chimera" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2004 | 0 | 1 | 1 |
| 2008 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2011 | 0 | 1 | 1 |
| 2012 | 0 | 1 | 1 |
| 2013 | 0 | 1 | 1 |
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Below are the most recent publications written about "Radiation Chimera" by people in Profiles.
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Harris DT, Badowski M. Long term human reconstitution and immune aging in NOD-Rag (-)-? chain (-) mice. Immunobiology. 2014 Feb; 219(2):131-7.
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Wong EB, Khan TN, Mohan C, Rahman ZS. The lupus-prone NZM2410/NZW strain-derived Sle1b sublocus alters the germinal center checkpoint in female mice in a B cell-intrinsic manner. J Immunol. 2012 Dec 15; 189(12):5667-81.
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Nikbakht N, Migone TS, Ward CP, Manser T. Cellular competition independent of BAFF/B lymphocyte stimulator results in low frequency of an autoreactive clonotype in mature polyclonal B cell compartments. J Immunol. 2011 Jul 1; 187(1):37-46.
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Li H, Matte-Martone C, Tan HS, Venkatesan S, McNiff J, Demetris AJ, Jain D, Lakkis F, Rothstein D, Shlomchik WD. Graft-versus-host disease is independent of innate signaling pathways triggered by pathogens in host hematopoietic cells. J Immunol. 2011 Jan 01; 186(1):230-41.
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Young FM, Campbell A, Emo KL, Jansson J, Wang PY, Jordan CT, Mullen CA. High-risk acute lymphoblastic leukemia cells with bcr-abl and INK4A/ARF mutations retain susceptibility to alloreactive T cells. Biol Blood Marrow Transplant. 2008 Jun; 14(6):622-30.
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Zhang L, Freedman NJ, Brian L, Peppel K. Graft-extrinsic cells predominate in vein graft arterialization. Arterioscler Thromb Vasc Biol. 2004 Mar; 24(3):470-6.