Thomas Jefferson University

Search Result Details

Back to Search Results
This page shows the details of why an item matched the keywords from your search.
Search Results
Eischen, Christine
One or more keywords matched the following items that are connected to Eischen, Christine
Item TypeName
Concept Tumor Suppressor Protein p53
Academic Article Phosphorylation of p53 serine 18 upregulates apoptosis to suppress Myc-induced tumorigenesis.
Academic Article A novel nuclear interactor of ARF and MDM2 (NIAM) that maintains chromosomal stability.
Academic Article The RING domain of Mdm2 can inhibit cell proliferation.
Academic Article The Mdm network and its regulation of p53 activities: a rheostat of cancer risk.
Academic Article Inactivation of p53 is insufficient to allow B cells and B-cell lymphomas to survive without Dicer.
Academic Article Decreased Mdm2 levels after DNA damage: antibody masking or protein degradation?
Academic Article Mdm2 promotes genetic instability and transformation independent of p53.
Academic Article Loss of miRNA biogenesis induces p19Arf-p53 signaling and senescence in primary cells.
Academic Article Pharmacologically Increasing Mdm2 Inhibits DNA Repair and Cooperates with Genotoxic Agents to Kill p53-Inactivated Ovarian Cancer Cells.
Academic Article The ARF tumor suppressor can promote the progression of some tumors.
Academic Article Loss of one allele of ARF rescues Mdm2 haploinsufficiency effects on apoptosis and lymphoma development.
Academic Article Potentiation of Carboplatin-Mediated DNA Damage by the Mdm2 Modulator Nutlin-3a in a Humanized Orthotopic Breast-to-Lung Metastatic Model.
Academic Article Mdm2 affects genome stability independent of p53.
Academic Article p53 and MDM2: antagonists or partners in crime?
Academic Article Mdm2 binds to Nbs1 at sites of DNA damage and regulates double strand break repair.
Academic Article Decreased Mdm2 expression inhibits tumor development and extends survival independent of Arf and dependent on p53.
Academic Article Murine double minute 2: p53-independent roads lead to genome instability or death.
Academic Article RABL6A, a novel RAB-like protein, controls centrosome amplification and chromosome instability in primary fibroblasts.
Academic Article Decoding the lncRNAome Across Diverse Cellular Stresses Reveals Core p53-effector Pan-cancer Suppressive lncRNAs.
Search Criteria
  • Tumor Suppressor Protein p53