Thomas Jefferson University

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Sigal, Luis
One or more keywords matched the following items that are connected to Sigal, Luis
Item TypeName
Concept Killer Cells, Natural
Concept NK Cell Lectin-Like Receptor Subfamily D
Concept Receptors, Natural Killer Cell
Concept NK Cell Lectin-Like Receptor Subfamily C
Concept NK Cell Lectin-Like Receptor Subfamily K
Academic Article Studying NK cell responses to ectromelia virus infections in mice.
Academic Article Age-dependent susceptibility to a viral disease due to decreased natural killer cell numbers and trafficking.
Academic Article A role for NKG2D in NK cell-mediated resistance to poxvirus disease.
Academic Article Memory CD8+ T cells specific for a single immunodominant or subdominant determinant induced by peptide-dendritic cell immunization protect from an acute lethal viral disease.
Academic Article The natural killer cell dysfunction of aged mice is due to the bone marrow stroma and is not restored by IL-15/IL-15Ra treatment.
Academic Article CD94 is essential for NK cell-mediated resistance to a lethal viral disease.
Academic Article Development and function of CD94-deficient natural killer cells.
Academic Article Enhanced NK-cell development and function in BCAP-deficient mice.
Academic Article Migratory Dendritic Cells, Group 1 Innate Lymphoid Cells, and Inflammatory Monocytes Collaborate to Recruit NK Cells to the Virus-Infected Lymph Node.
Academic Article Chronic Lymphocytic Choriomeningitis infection causes susceptibility to mousepox and impairs Natural Killer cell maturation and function.
Academic Article a2ß1 Integrin Is Required for Optimal NK Cell Proliferation during Viral Infection but Not for Acquisition of Effector Functions or NK Cell-Mediated Virus Control.
Academic Article Declined miR-181a-5p expression is associated with impaired natural killer cell development and function with aging.
Academic Article Viral infection modulates Qa-1b in infected and bystander cells to properly direct NK cell killing.
Academic Article Resistance to lethal ectromelia virus infection requires Type I interferon receptor in natural killer cells and monocytes but not in adaptive immune or parenchymal cells.
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  • Killer Cells Natural