Thomas Jefferson University

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Alnemri, Emad
One or more keywords matched the following items that are connected to Alnemri, Emad
Item TypeName
Concept Calcium-Binding Proteins
Concept Carrier Proteins
Concept DNA-Binding Proteins
Concept Protein Binding
Academic Article Functional consequences of a germline mutation in the leucine-rich repeat domain of NLRP3 identified in an atypical autoinflammatory disorder.
Academic Article Involvement of the AIM2, NLRC4, and NLRP3 inflammasomes in caspase-1 activation by Listeria monocytogenes.
Academic Article Critical roles of ASC inflammasomes in caspase-1 activation and host innate resistance to Streptococcus pneumoniae infection.
Academic Article Rapid kinetics of tBid-induced cytochrome c and Smac/DIABLO release and mitochondrial depolarization.
Academic Article sickle, a novel Drosophila death gene in the reaper/hid/grim region, encodes an IAP-inhibitory protein.
Academic Article CARD-8 protein, a new CARD family member that regulates caspase-1 activation and apoptosis.
Academic Article Caspase-2 induces apoptosis by releasing proapoptotic proteins from mitochondria.
Academic Article The PYRIN-CARD protein ASC is an activating adaptor for caspase-1.
Academic Article Role of Smac in human leukaemic cell apoptosis and proliferation.
Academic Article Involvement of proapoptotic molecules Bax and Bak in tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced mitochondrial disruption and apoptosis: differential regulation of cytochrome c and Smac/DIABLO release.
Academic Article The polypeptide chain-releasing factor GSPT1/eRF3 is proteolytically processed into an IAP-binding protein.
Academic Article Tim50, a component of the mitochondrial translocator, regulates mitochondrial integrity and cell death.
Academic Article Pyrin activates the ASC pyroptosome in response to engagement by autoinflammatory PSTPIP1 mutants.
Academic Article X-linked and cellular IAPs modulate the stability of C-RAF kinase and cell motility.
Academic Article AIM2 activates the inflammasome and cell death in response to cytoplasmic DNA.
Academic Article THAP5 is a human cardiac-specific inhibitor of cell cycle that is cleaved by the proapoptotic Omi/HtrA2 protease during cell death.
Academic Article Cutting edge: NF-kappaB activating pattern recognition and cytokine receptors license NLRP3 inflammasome activation by regulating NLRP3 expression.
Academic Article Cutting edge: TLR signaling licenses IRAK1 for rapid activation of the NLRP3 inflammasome.
Academic Article The mitochondrial antiviral protein MAVS associates with NLRP3 and regulates its inflammasome activity.
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  • Protein Binding