"Hyperalgesia" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An increased sensation of pain or discomfort produced by mimimally noxious stimuli due to damage to soft tissue containing NOCICEPTORS or injury to a peripheral nerve.
| Descriptor ID |
D006930
|
| MeSH Number(s) |
C10.597.751.791.400 C23.888.592.763.770.400
|
| Concept/Terms |
Hyperalgesia- Hyperalgesia
- Hyperalgesias
- Hyperalgesic Sensations
Mechanical Allodynia- Mechanical Allodynia
- Allodynias, Mechanical
- Hyperalgesia, Mechanical
- Hyperalgesias, Mechanical
- Mechanical Hyperalgesia
- Allodynia, Mechanical
Tactile Allodynia- Tactile Allodynia
- Allodynias, Tactile
- Hyperalgesia, Tactile
- Hyperalgesias, Tactile
- Allodynia, Tactile
Hyperalgesia, Thermal- Hyperalgesia, Thermal
- Hyperalgesias, Thermal
- Thermal Hyperalgesia
- Thermal Allodynia
- Allodynia, Thermal
- Allodynias, Thermal
|
Below are MeSH descriptors whose meaning is more general than "Hyperalgesia".
Below are MeSH descriptors whose meaning is more specific than "Hyperalgesia".
This graph shows the total number of publications written about "Hyperalgesia" by people in this website by year, and whether "Hyperalgesia" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2005 | 2 | 0 | 2 |
| 2007 | 1 | 0 | 1 |
| 2009 | 1 | 0 | 1 |
| 2010 | 0 | 1 | 1 |
| 2011 | 1 | 0 | 1 |
| 2012 | 2 | 0 | 2 |
| 2016 | 1 | 1 | 2 |
| 2017 | 1 | 0 | 1 |
| 2018 | 0 | 1 | 1 |
| 2019 | 1 | 2 | 3 |
| 2023 | 0 | 2 | 2 |
| 2024 | 1 | 3 | 4 |
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click here.
Below are the most recent publications written about "Hyperalgesia" by people in Profiles.
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Ippolito M, Hayduk SA, Kinney W, Brenneman DE, Ward SJ. KLS-13019, a Novel Structural Analogue of Cannabidiol and GPR55 Receptor Antagonist, Prevents and Reverses Chemotherapy-Induced Peripheral Neuropathy in Rats. J Pharmacol Exp Ther. 2024 10 18; 391(2):231-240.
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David ET, Yousuf MS, Mei HR, Jain A, Krishnagiri S, Elahi H, Venkatesan R, Srikanth KD, Dussor G, Dalva MB, Price TJ. ephrin-B2 promotes nociceptive plasticity and hyperalgesic priming through EphB2-MNK-eIF4E signaling in both mice and humans. Pharmacol Res. 2024 08; 206:107284.
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Li H, Ward SJ. Paclitaxel-Associated Mechanical Sensitivity and Neuroinflammation Are Sex-, Time-, and Site-Specific and Prevented through Cannabigerol Administration in C57Bl/6 Mice. Int J Mol Sci. 2024 Apr 12; 25(8).
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Brenneman DE, Kinney WA, McDonnell ME, Ippolito MJ, Ward SJ. Knockdown siRNA Targeting GPR55 Reveals Significant Differences Between the Anti-inflammatory Actions of KLS-13019 and Cannabidiol. J Mol Neurosci. 2024 Apr 11; 74(2):41.
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Ahmed AK, Zhuo J, Gullapalli RP, Jiang L, Keaser ML, Greenspan JD, Chen C, Miller TR, Melhem ER, Sansur CA, Eisenberg HM, Gandhi D. Focused Ultrasound Central Lateral Thalamotomy for the Treatment of Refractory Neuropathic Pain: Phase I Trial. Neurosurgery. 2024 Apr 01; 94(4):690-699.
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Vincent KF, Bundock J, Dona CPG, Chenna SS, Mohanty S, Saini C, Hong J, Albert TJ, Dahia CL. Loss of lumbar disc height with age and its impact on pain and sensitivity associated behaviors in mice. Eur Spine J. 2023 03; 32(3):848-858.
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Brenneman DE, Kinney WA, Ward SJ. Knockdown siRNA Targeting the Mitochondrial Sodium-Calcium Exchanger-1 Inhibits the Protective Effects of Two Cannabinoids Against Acute Paclitaxel Toxicity. J Mol Neurosci. 2019 Aug; 68(4):603-619.
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Vincent K, Mohanty S, Pinelli R, Bonavita R, Pricop P, Albert TJ, Dahia CL. Aging of mouse intervertebral disc and association with back pain. Bone. 2019 06; 123:246-259.
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Young WB, Ivan Lopez J, Rothrock JF, Orejudos A, Manack Adams A, Lipton RB, Blumenfeld AM. Effects of onabotulinumtoxinA treatment in patients with and without allodynia: results of the COMPEL study. J Headache Pain. 2019 Jan 22; 20(1):10.
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Li H, Kong W, Chambers CR, Yu D, Ganea D, Tuma RF, Ward SJ. The non-psychoactive phytocannabinoid cannabidiol (CBD) attenuates pro-inflammatory mediators, T cell infiltration, and thermal sensitivity following spinal cord injury in mice. Cell Immunol. 2018 07; 329:1-9.