Retinoic Acid Receptor alpha
"Retinoic Acid Receptor alpha" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A nuclear receptor protein which belongs to a family of transcription regulators of ligand-responsive regulatory proteins which include steroid hormone receptors. In addition to a C-terminal ligand-binding domain, these nuclear receptors contain a highly-conserved N-terminal zinc-finger domain that mediates binding ligand-responsive elements.
| Descriptor ID |
D000072482
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| MeSH Number(s) |
D12.776.826.701.250 D12.776.930.775.250
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| Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Retinoic Acid Receptor alpha".
Below are MeSH descriptors whose meaning is more specific than "Retinoic Acid Receptor alpha".
This graph shows the total number of publications written about "Retinoic Acid Receptor alpha" by people in this website by year, and whether "Retinoic Acid Receptor alpha" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 1 | 1 |
| 2017 | 0 | 1 | 1 |
| 2019 | 1 | 0 | 1 |
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Below are the most recent publications written about "Retinoic Acid Receptor alpha" by people in Profiles.
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Wang Q, Thome R, Rostami A, Ma CG, Zhang GX. The selective retinoic acid receptor-a agonist AM580 fails to control autoimmune neuroinflammation. Cell Mol Immunol. 2019 08; 16(8):727-729.
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Yao PL, Chen L, Dobrzanski TP, Zhu B, Kang BH, M?ller R, Gonzalez FJ, Peters JM. Peroxisome proliferator-activated receptor-?/d inhibits human neuroblastoma cell tumorigenesis by inducing p53- and SOX2-mediated cell differentiation. Mol Carcinog. 2017 05; 56(5):1472-1483.
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Katsetos CD, Stadnicka I, Boyd JC, Ehya H, Zheng S, Soprano CM, Cooper HS, Patchefsky AS, Soprano DR, Soprano KJ. Cellular distribution of retinoic acid receptor-alpha protein in serous adenocarcinomas of ovarian, tubal, and peritoneal origin: comparison with estrogen receptor status. Am J Pathol. 1998 Aug; 153(2):469-80.